Diphenidine is a synthetic dissociative of the diarylethylamine class — a structural departure from the arylcyclohexylamine family (ketamine, PCP, DCK) in that it contains two phenyl rings rather than one, connected through an ethylamine chain rather than a cyclohexyl ring. This structural distinction gives diphenidine a qualitatively different pharmacological and experiential character: community members and researchers note that diphenidine produces a more dysphoric, "dark," or unsettling dissociation compared to arylcyclohexylamines, with less of the warmth or euphoria associated with compounds like ketamine or 3-HO-PCP.
Diphenidine acts as an NMDA receptor antagonist, producing dissociation, analgesia, and at higher doses, full dissociative anesthesia. Unlike arylcyclohexylamines, it may have less dopaminergic activity, contributing to a different "flavor" of dissociation. The diarylethylamine class is associated with what community members describe as a colder, more clinical dissociation that can veer into uncomfortable territory at higher doses. Community and anecdotal evidence suggests diphenidine has a distinct toxicity profile from arylcyclohexylamines and that vaporizing diphenidine yields a substantially different and often preferable experience compared to ephenidine, its ethyl-substituted analog.
Diphenidine was widely available during the 2010s research chemical era and generated significant community discussion, including accounts of its use for analgesia in chronic pain contexts.
Safety at a Glance
High Risk- Approach as an Under-Characterized Substance
- Diphenidine lacks the extensive community experience base of ketamine or MXE. This uncertainty should itself be treat...
- Toxicity: Acute Toxicity Diphenidine's toxicity profile is not well characterized by formal research. As an NMDA antagonist, th...
- Dangerous with: Acetylfentanyl, Buprenorphine, Codeine, Desomorphine (+15 more)
- Overdose risk: Overdose on Diphenidine can range from unpleasant to life-threatening depending on the dose, rout...
If someone is in crisis, call 911 or Poison Control: 1-800-222-1222
Dosage
oral
rectal
smoked
Duration
oral
Total: 2 hrs – 5 hrsrectal
Total: 5 hrs – 7 hrssmoked
Total: 1 hrs – 3 hrsHow It Feels
The onset is slow and carries an immediate undercurrent of unease, as though the compound is entering through a back door you did not know existed. Thirty to forty-five minutes pass before the first clear signal arrives: a heaviness in the body, a dulling of sensation, and a strange darkening of the mental atmosphere. This is not a bright dissociative. From the very beginning, Diphenidine has a subterranean quality, a sense of descent rather than ascent, as though you are being slowly lowered into a well.
The come-up continues for an hour or more, and the confusion builds in layers. Each layer removes something: first the sharpness of vision, then the coherence of thought, then the sense of time's forward motion, then the feeling of being located in a particular place. What replaces these is a dark, amorphous space that is neither pleasant nor frightening — simply alien, a place where the usual coordinates of experience have been quietly removed. Sound becomes a distant rumble, as though you are hearing the world through several feet of earth. Conversations, if anyone is speaking, arrive as meaningless rhythmic patterns. The body is profoundly numb, almost absent.
The peak is a place of deep, amnesic dissociation. This is one of the most reliably hole-producing compounds in the diarylethylamine family, but the hole it produces is not the crystalline architecture of ketamine or the warm caverns of MXE — it is dark, formless, and vast, a place where consciousness persists but has nothing to attach to. There may be fragments of imagery — fleeting geometric forms, faces that dissolve on inspection — but they do not cohere into narrative or meaning. Time does not pass; it simply is not present. The emotional register is flatlined, neither blissful nor terrifying, just deeply, fundamentally blank. Memory formation is severely impaired; you may spend an hour in this space and retain only a few seconds of it.
The return is disorienting and extremely protracted. Baseline may be eight, ten, even twelve hours away. You surface through layers of confusion, each one restoring a single dimension of ordinary experience: first location, then time, then language, then the feeling of having a body. The amnesia extends into the comedown — hours may pass with no recollection. The morning after carries a heavy, depleted fog, a sense of having been somewhere very far away and having brought nothing back except exhaustion.
Subjective Effects
The effects listed below are based on the Subjective Effect Index (SEI), an open research literature based on anecdotal reports and personal analyses. They should be viewed with a healthy degree of skepticism. These effects will not necessarily occur in a predictable or reliable manner, although higher doses are more liable to induce the full spectrum of effects.
Physical Effects
Physical(16)
- Changes in felt bodily form— Changes in felt bodily form is the experience of one's body feeling as though it has altered its phy...
- Dizziness— A sensation of spinning, swaying, or lightheadedness that impairs balance and spatial orientation, o...
- Gait alteration— Gait alteration is a noticeable change in the way a person walks and moves through their environment...
- Increased heart rate— A noticeable acceleration of heartbeat that can range from a subtle awareness of one's pulse to a fo...
- Increased libido— A marked enhancement of sexual desire, arousal, and sensitivity to erotic stimuli that can range fro...
- Increased salivation— Increased salivation (hypersalivation or sialorrhea) is the excessive production of saliva beyond wh...
- Motor control loss— A distinct decrease in the ability to control one's physical body with precision, balance, and coord...
- Nausea— An uncomfortable sensation of queasiness and stomach discomfort that may or may not lead to vomiting...
- Orgasm suppression— Orgasm suppression (anorgasmia) is the difficulty or complete inability to achieve orgasm despite ad...
- Perception of bodily lightness— Perception of bodily lightness is the subjective feeling that one's body has become dramatically lig...
- Physical autonomy— Physical autonomy is the experience of one's body performing actions — from simple tasks like walkin...
- Physical euphoria— An intensely pleasurable bodily sensation that can manifest as waves of warmth, tingling electricity...
- Respiratory depression— A dangerous slowing and shallowing of breathing that can progress from barely noticeable reductions ...
- Sedation— A state of deep physical and mental calming that manifests as a progressive desire to remain still, ...
- Spatial disorientation— Spatial disorientation is the inability to accurately perceive one's position or orientation within ...
- Stimulation— A state of heightened physical and mental energy characterized by increased wakefulness, elevated mo...
Tactile(1)
- Tactile suppression— A progressive decrease in the ability to feel physical touch, ranging from mild numbness to complete...
Cognitive & Perceptual Effects
Visual(11)
- Double vision— The visual experience of seeing a single object as two separate, overlapping images, similar to cros...
- Environmental cubism— A visual distortion in which the environment and objects within it appear fragmented into geometric,...
- Frame rate suppression— Perception of visual motion as choppy discrete frames rather than smooth continuous flow, resembling...
- Geometry— The experience of perceiving complex, ever-shifting geometric patterns superimposed over the visual ...
- Internal hallucination— Vivid, detailed visual experiences perceived within an imagined mental landscape that can only be se...
- Perspective distortions— Distortion of perceived depth, distance, and size of real objects, making things appear closer, furt...
- Perspective hallucination— A hallucinatory phenomenon in which the observer's visual perspective shifts from the normal first-p...
- Scenery slicing— The visual field fractures into distinct, cleanly cut sections that slowly drift apart from their or...
- Settings, sceneries, and landscapes— The perceived environment in which hallucinatory experiences take place, ranging from recognizable l...
- Visual acuity suppression— Vision becomes blurred, indistinct, and out of focus, as though looking through a smudged lens. Fine...
- Visual disconnection— A dissociative visual effect involving a progressive detachment from visual perception, ranging from...
Cognitive(21)
- Amnesia— A complete or partial inability to form new memories or recall existing ones during and after substa...
- Analysis suppression— Analysis suppression is a cognitive impairment in which the capacity for logical reasoning, critical...
- Anxiety— Intense feelings of apprehension, worry, and dread that can range from a subtle background unease to...
- Anxiety suppression— A partial to complete suppression of anxiety and general unease, producing a calm, relaxed mental st...
- Cognitive euphoria— A cognitive and emotional state of intense well-being, elation, happiness, and joy that manifests as...
- Compulsive redosing— An overwhelming, difficult-to-resist urge to continuously take more of a substance in order to maint...
- Conceptual thinking— A shift in the nature of thought from verbal, linear sentence structures to intuitive, non-linguisti...
- Confusion— An impairment of abstract thinking marked by a persistent inability to grasp or comprehend concepts ...
- Creativity enhancement— An increase in the ability to imagine new ideas, overcome creative blocks, think about existing conc...
- Deja vu— Intense, often prolonged sensation of having already experienced the current moment, common with psy...
- Depersonalization— A detachment from one's own sense of self, body, or mental processes, as if observing oneself from o...
- Depression— A persistent state of low mood, emotional numbness, hopelessness, and diminished interest or pleasur...
- Derealization— A perceptual disturbance in which the external world feels profoundly unreal, dreamlike, or artifici...
- Disinhibition— A marked reduction in social inhibitions, self-consciousness, and behavioral restraint that manifest...
- Dream potentiation— Enhanced dream vividness, complexity, and recall, often occurring as REM rebound after discontinuing...
- Ego inflation— Grandiose overconfidence and inflated self-importance, opposite of ego death, commonly produced by s...
- Introspection— An enhanced state of self-reflective awareness in which one feels drawn to examine their own thought...
- Memory suppression— A dose-dependent inhibition of one's ability to access and utilize short-term and long-term memory, ...
- Paranoia— Irrational suspicion and belief that others are watching, plotting against, or intending harm toward...
- Thought deceleration— The experience of thoughts occurring at a markedly reduced pace, as if the mind has been placed into...
- Time distortion— Subjective perception of time becomes dramatically altered — minutes may feel like hours, or hours p...
Auditory(5)
- Auditory distortion— Auditory distortion is the experience of sounds becoming warped, pitch-shifted, flanged, or otherwis...
- Auditory enhancement— Auditory enhancement is a heightened sensitivity and appreciation of sound in which music, voices, a...
- Auditory hallucination— Auditory hallucination is the perception of sounds that have no external source — hearing music, voi...
- Auditory misinterpretation— Auditory misinterpretation is the brief, spontaneous misidentification of real sounds as entirely di...
- Auditory suppression— A dampening of auditory perception in which sounds become muffled, distant, and reduced in both volu...
Multi-sensory(2)
- Memory replays— Memory replays are vivid, multisensory re-experiences of past events that go far beyond normal recal...
- Scenarios and plots— Scenarios and plots are the narrative structures that emerge within hallucinatory states — coherent ...
Transpersonal(1)
- Ego death— A profound dissolution of the sense of self in which personal identity, memories, and the boundary b...
Pharmacology
Mechanism of Action
Diphenidine acts as a non-competitive NMDA receptor antagonist — blocking the NMDA receptor ion channel and disrupting glutamatergic neurotransmission. The pharmacological mechanism is shared with arylcyclohexylamines but the receptor binding kinetics and selectivity may differ given the substantial structural differences.
Structural Distinctives of the Diarylethylamine Class
Diarylethylamines like diphenidine lack the cyclohexyl ring that characterizes arylcyclohexylamines. This structural difference produces:
- Potentially different NMDA receptor binding kinetics (on/off rates, trapping behavior)
- Altered selectivity across receptor subtypes
- Different metabolic pathways producing a different metabolite profile
- A qualitatively different experiential character as reported by community members
Dopaminergic Activity
Unlike PCP-class compounds, diphenidine likely has less significant dopamine reuptake inhibitory activity, contributing to the less stimulant and more sedating quality noted by community members.
Potency and Duration
Diphenidine is less potent than most arylcyclohexylamines on a per-weight basis, with active doses typically in the 25–75 mg range depending on route. Duration of action extends to several hours. Community accounts (including comparative reports between diphenidine and ephenidine via vaporization) indicate meaningful pharmacokinetic differences between administration routes.
Detection Methods
Standard Drug Panel Inclusion
Diphenidine is NOT included on standard drug screening panels. It does not cross-react with PCP, ketamine, or any other immunoassay class used in conventional drug testing. Standard 5-panel, 10-panel, and extended panels will not detect this compound. The diarylethylamine structure is sufficiently distinct from the arylcyclohexylamines targeted by PCP screens.
Urine Detection
Diphenidine is detectable in urine for approximately 2 to 5 days after a single dose. The compound undergoes hepatic metabolism via N-dealkylation, hydroxylation, and oxidation, producing multiple metabolites. Specific metabolic pathways have been characterized through in vitro studies and case reports. LC-MS/MS methods targeting diphenidine and its metabolites are required for reliable detection.
Blood and Serum Detection
Blood detection windows are approximately 1 to 3 days. Active blood concentrations in forensic cases range from 10 to 500 ng/mL. Fatal cases have been reported with blood concentrations in the hundreds of nanograms-per-milliliter range. The compound has a duration of action of 3 to 5 hours, indicating moderate metabolic stability.
Hair Follicle Detection
Hair analysis for diphenidine has been performed in forensic contexts using LC-MS/MS methods. The lipophilic, basic nature of the compound supports hair incorporation, with detection windows up to 90 days.
Confirmatory Methods
LC-MS/MS provides definitive identification and quantification. The diarylethylamine structure produces characteristic fragmentation patterns in mass spectrometry. GC-MS is also applicable. Several forensic methods have been published and validated.
Reagent Testing
The Marquis reagent produces no reaction or a faint yellow color. The Mandelin reagent may yield a brown response. The Mecke reagent may produce a yellow-green color. These reagent profiles are insufficient for definitive identification among dissociative compounds. Fentanyl test strips should be used on any material from unregulated sources.
Interactions
| Substance | Status | Note |
|---|---|---|
| Acetylfentanyl | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Buprenorphine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Codeine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Desomorphine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Dextropropoxyphene | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Dihydrocodeine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Ethylmorphine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Fentanyl | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Heroin | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Hydrocodone | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Hydromorphone | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Kratom | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Methadone | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Morphine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Naloxone | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| O-Desmethyltramadol | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Oxycodone | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Oxymorphone | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| Pethidine | Dangerous | Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration |
| 1,4-Butanediol | Caution | Both cause CNS depression; increased risk of vomiting, unconsciousness, and respiratory depression |
| 2-Fluorodeschloroketamine | Caution | Compounding dissociative effects can cause confusion, mania, and loss of motor control |
| 2M2B | Caution | Both cause CNS depression; increased risk of vomiting, unconsciousness, and respiratory depression |
| 3-Cl-PCP | Caution | Compounding dissociative effects can cause confusion, mania, and loss of motor control |
| 3-HO-PCE | Caution | Compounding dissociative effects can cause confusion, mania, and loss of motor control |
| 1,3-Butanediol | Low Risk & Synergy | Produces unique synergistic effects; often combined |
| 1B-LSD | Low Risk & Synergy | Produces unique synergistic effects; often combined |
| 1cP-AL-LAD | Low Risk & Synergy | Produces unique synergistic effects; often combined |
| 1cP-LSD | Low Risk & Synergy | Produces unique synergistic effects; often combined |
| 1cP-MiPLA | Low Risk & Synergy | Produces unique synergistic effects; often combined |
History
Development and Research Background
Diphenidine and related diarylethylamines were synthesized and investigated in the mid-20th century as part of SAR exploration of NMDA antagonist compounds. Unlike arylcyclohexylamines, the diarylethylamine scaffold was less heavily developed for therapeutic purposes, and these compounds received less systematic pharmacological characterization.
Research Chemical Era
Diphenidine emerged prominently in the research chemical market during the mid-2010s, particularly in European markets where it served as a novel dissociative experience for users interested in the class. It was often available alongside ephenidine (its ethyl analog) and compared favorably by many community members seeking specific experiential qualities.
Class Significance
Diphenidine and ephenidine represent the diarylethylamine class's most prominent community engagement. Their distinct pharmacological character from arylcyclohexylamines — and the documented experiential differences compared to ketamine-type compounds — contributed to the broader understanding within harm reduction communities that "dissociative" encompasses pharmacologically diverse compounds with meaningfully different risk and experience profiles.
Harm Reduction
Approach as an Under-Characterized Substance
Diphenidine lacks the extensive community experience base of ketamine or MXE. This uncertainty should itself be treated as a risk factor requiring extra caution.
Start Low — Active Dose Range is Wide
Active doses range from 25 mg to 75 mg or more, with a wide range of individual sensitivity. Begin at 25 mg oral and assess fully before any consideration of redosing.
Physical Safety Environment
Diphenidine's motor impairment at dissociative doses is substantial. Ensure you cannot fall, and that the environment is clear of hazards. Have a sober companion for higher-dose explorations.
Monitor for Dysphoria
Community accounts suggest diphenidine is more prone to producing dysphoric, uncomfortable experiences at higher doses than arylcyclohexylamines. If a session becomes intensely negative, changing the environment (music, lighting, location) and focusing on slow breathing may help. Having benzodiazepines available as a safety net is a community-standard practice.
No CNS Depressant Combinations
Alcohol, opioids, benzodiazepines, and GHB increase respiratory depression risk. Use alone or in combination only with awareness of this risk.
Toxicity & Safety
Acute Toxicity
Diphenidine's toxicity profile is not well characterized by formal research. As an NMDA antagonist, the primary acute risks include: respiratory depression at very high doses (especially combined with other CNS depressants), cardiovascular effects, and profound motor and cognitive impairment that creates injury risk.
Psychological Toxicity
The "dark" or dysphoric quality noted by many users at higher doses suggests a higher rate of difficult or overwhelming experiences compared to more euphoric dissociatives. Acute psychological crisis, paranoia, and panic are documented risks.
Class-Specific Uncertainty
As a diarylethylamine rather than arylcyclohexylamine, diphenidine may have a distinct bladder toxicity profile — the mechanism of ketamine bladder toxicity involves specific metabolites, and the metabolite profile of diarylethylamines differs. Whether this means a higher or lower urological risk is not established.
Drug Interactions
CNS depressants (alcohol, opioids, benzodiazepines) increase respiratory depression risk. The combination of diphenidine with other substances should be approached with the same caution as any NMDA antagonist.
Psychological Dependence
As with other dissociatives, psychological dependence with compulsive use patterns is a risk, particularly given the initially novel experience character.
Addiction Potential
moderately addictive with a high potential for abuse
Overdose Information
Overdose on Diphenidine can range from unpleasant to life-threatening depending on the dose, route, and whether other substances are involved.
Signs of overdose: Severe nystagmus, complete unresponsiveness, vomiting (aspiration risk while unconscious), severely depressed breathing, seizures, extremely elevated heart rate or blood pressure.
Critical dangers:
- Respiratory depression: Particularly when combined with other depressants
- Aspiration: Loss of protective reflexes combined with nausea creates choking risk
- Hyperthermia or hypothermia: Impaired thermoregulation at high doses
Emergency response: Place the person in the recovery position. Monitor breathing. Call emergency services if breathing is slow, shallow, or irregular; if the person is unresponsive to stimulation; or if seizures occur. Be honest with medical personnel about what was consumed — they are there to help, not to judge.
Dangerous Interactions
The combinations listed below may be life-threatening. Independent research should always be conducted to ensure safety when combining substances.
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Both cause respiratory depression and unconsciousness; vomiting while dissociated risks aspiration
Tolerance
| Full | develops with prolonged and repeated use |
| Half | 3 - 7 days |
| Zero | 1 - 2 weeks |
Cross-tolerances
Legal Status
Diphenidine is currently a legal grey area drug worldwide and is easily accessible through the use of online research chemical vendors. However, this does not guarantee anyone to be immune from legal prosecution should they be found in possession of this substance as the legality is likely to vary from country to country.
Canada:** As of March 2016, MT-45 and its analogues, one of which being Diphenidine, are Schedule I controlled substances. Possession without legal authority can result in maximum 7 years imprisonment. Only those with a law enforcement agency, person with an exemption permit or institutions with Minister's authorization may possess the drug.
Switzerland:** Diphenidine is a controlled substance specifically named under Verzeichnis E.
Turkey:** Diphenidine is a classed as drug and is illegal to possess, produce, supply, or import.
United Kingdom:** It is illegal to produce, supply, or import this drug under the Psychoactive Substance Act, which came into effect on May 26th, 2016.
United States:** Diphenidine is a schedule II substance and is illegal without a prescription.
Responsible use
Dissociative
Diarylethylamine
Diphenidine (Wikipedia)
Diphenidine (Isomer Design)
Community
The Big & Dandy Diphenidine Thread (Bluelight)
Wallach, J., Kang, H., Colestock, T., Morris, H., Bortolotto, Z. A., Collingridge, G. L., ... & Adejare, A. (2016). Pharmacological investigations of the dissociative ‘legal highs’ diphenidine, methoxphenidine and analogues. PLoS One, 11(6), e0157021. https://doi.org/10.1371/journal.pone.0157021
Morris, H., & Wallach, J. (2014). From PCP to MXE: A comprehensive review of the non-medical use of dissociative drugs. Drug Testing and Analysis, 6(7–8), 614–632. https://doi.org/10.1002/dta.1620
Experience Reports (1)
Tips (5)
If insufflating Diphenidine, start with a small bump and wait 15-20 minutes to assess effects before taking more. Onset via insufflation is faster than oral, but full effects still take time to manifest.
The moreish quality of many dissociatives including Diphenidine makes compulsive redosing common. Pre-measure your intended dose and put the rest away. A timed lockbox can help prevent impulsive additional doses.
Weigh your dose of Diphenidine with a milligram scale. Dissociative dose-response curves are steep. The difference between a pleasant floaty experience and complete dissociation from reality can be 50mg or less.
Do not combine Diphenidine with other CNS depressants (alcohol, opioids, benzodiazepines). Dissociatives already depress breathing and cardiovascular function. Adding other depressants creates dangerous respiratory depression.
Tolerance to Diphenidine builds with regular use. Taking breaks of at least 1-2 weeks between sessions helps prevent tolerance escalation and reduces the risk of bladder and cognitive damage from frequent use.
See Also
References (4)
- Ketamine as a rapid antidepressant — Berman et al. Biological Psychiatry (2000)paper
- PubChem: Diphenidine
PubChem compound page for Diphenidine (CID: 206666)
pubchem - Diphenidine - TripSit Factsheet
TripSit factsheet for Diphenidine
tripsit - Diphenidine - Wikipedia
Wikipedia article on Diphenidine
wikipedia